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Leg Ulcers

In venous leg ulcers, the incompetency of venous valves means there may be backflow in the veins of the lower leg which results in venous hypertension. This can cause fluid to accumulate in the tissues, developing oedema.

Brown haemosiderin staining is caused by the breakdown of red blood cells which become trapped in the skin. Induration occurs from fibrosis of the subcutaneous layer which may result in a classic ‘champagne leg’. Other venous symptoms are varicose eczema, oedema, ankle flare (distended veins in medial ankle area) pain (with relief on elevating the limb), varicose veins, ulcers in gaiter or malleolus regions.

Risk factors of venous ulceration are: DVT, varicose veins, swollen oedematous legs, multiple pregnancies, lower leg fracture, thrombophlebitis, previous leg ulceration, previous vascular or orthopaedic surgery.

Arterial signs include: reduced or absent pedal pulses, history of intermittent claudication, reduced ABPI, deep punched out ulcers on toes, heels or foot, necrosis or gangrene, loss of hair to the limb, shiny, pale hairless skin on shin, dusky coloured foot, cool to touch, thickened toe nails, pain in feet and blanching when elevated, delayed capillary refill.

Risk factors for arterial ulcers include ischaemic heart disease, smoking, hypertension, diabetes mellitus, TIA/CVA, MI or angina, rheumatoid arthritis or previous arterial surgery.

Compression bandaging used in treatment of venous leg ulcers is designed to aid venous return. Laplace’s law is that the pressure applied to the limb is determined by the width of the bandage, the degree of overlap and degree and technique of stretch applied (usually both 50%) but this is dependent on the ankle being smaller than the calf. Shortstretch bandaging is different in that it is applied at 100% stretch.

  • 80% of leg ulcers are venous
  • Venous ulcers are caused by chronic venous insufficiency; pooling in the leg leads to venous congestion, leading to fluid being forced out of the vessel and into the surrounding tissues as oedema.
  • Erythrocytes leaking through into the tissue can stain the leg, a symptom of chronic venous disease.
  • Oedema can also be caused or exacerbated by renal or cardiac conditions, and therefore their involvement needs to be ascertained as part of care planning.
 

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Care for Burns

  • Apply cool (around 15ºC) water for 20mins (even up to 3 hours post burn, this treatment can still be effective), this serves to reduce vasoconstriction, inflammation and decreased oedema.
  • Do not apply ice, as this can promote vasoconstriction and even increase depth of burn.
    Chemical burns need to be irrigated with copious amounts of water.
  • When wound is cooled, apply a clean dressing, cling film is OK as first aid as it doesn’t shed fibres into the wound, and is easy to remove as well as being transparent so the wound can be seen without first removing the dressing. Swelling may cause constriction, so if applying cling film, do not wrap around wound, but rather lay across wound and secure.
  • Obtain history of injury, including rough temperature of the object and length of exposure time.
  • To aid healing process, apply sterile antimicrobial dressings using aseptic technique to act as barrier to bacteria, and maintain a moist environment.
  • Wounds may deepen in first 48 hours.
  • Remove dead tissue from wound to prevent this harbouring bacteria. Only burst blisters if absolutely necessary.

Edwards, J. (2012) Burn wound and scar management Nursing in Practice 64 41-44

 
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Posted by on March 5, 2012 in Acute care, Wounds

 

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Care of the Diabetic Foot

Diabetes is a condition with far-reaching consequences. As the population ages, more diabetes-related conditions will occur.

Diabetes is the root cause of the majority of non-traumatic limb amputations, and therefore diabetic foot problems need to be treated as an emergency, according to NICE guidelines.

Diabetic foot problems can include neuropathy, charcot arthropathy (or other deformity), gangrene, ulcers, osteomyelitis, peripheral arterial disease, or infection.

For care of diabetic foot problesm, the multidisciplinary team in an acute setting will comprise: tissue viability nurse, diabetologist, relevant surgeon, diabetes specialist nurse, podiatrist, and possibly a physiotherapist. The aim of the MDT is primarily mobilisation.

Patients with diabetes-related foot problems need to be referred to the responsible MDT within 24 hours of admission. this team will then assess and initiate treatment of  the patient’s underlying diabetes, assess and coordinate care for the presenting foot problems, (the assessment is to include a vascular assessment). Infection also needs to be treated immediately; a swab may be taken, but depending on the stage of infection, it is likely that antibiotics are started before results return from the lab. At this point the patient will be assessed with regard to their need for orthotics or other interventions to protect the feet and revent/reduce future problems where possible. The MDT will also begin to consider discharge, so there is a workable plan in place at the right time.

At all stages, the patient must be kept in the loop with discussions and must be a part of the decision-making process. Patient education and empowerment is paramount, as is effective communication with the primary care team taking over care (if necessary).

 
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Posted by on February 27, 2012 in Diabetes, Wounds

 

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Swabs

When swabbing wounds the Levine technique is more effective than the Z technique to determine colonisation/infection of wounds.

The Z technique involves sweeping the swab in a Z across the wound bed (avoiding the edges). The Levine technique samples more organisms because by rotating the swab over a small area (1cm square) it picks up more organisms from the surface as well as just below the wound surface.

 
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Posted by on February 26, 2012 in Wounds

 

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